Sometimes a Systemic Yeast Infection can have light symptoms, and because of this they're dismissed as nothing to concerned about. The problem is, this allows the yeast to enter the bloodstream where a lot of different problems can eventuate and can even become a life-threatening situation.

The body naturally has a certain amount of yeast that lives in the body in small quantities and actually help the body, without doing any harm to it. There are also different types of microorganisms inside the body that are there to keep the yeast in check so an infection does not occur.

On-going stress attacks, poor choices of food, normal pregnancy changes, immune system deficiency diseases, antibiotic medications and other disease may abolish the tiny microorganisms and thereby allow the Systemic Yeast Infection Symptoms to rage.

The Systemic Yeast Infection Symptoms can differ from each individual, but here a few of the most usual ones:

Heavy sense of exhaustion and unusual run down feelings may arise.

1. The feelings of sensory disturbances, unusual muscle aches and pains, continual headaches, constant dizziness, and complaints of the sufferering of persistent tiredness are signs of a person with Systemic Yeast Infection.

2. Unusual or sudden sensitivity to chemicals or new food allergies.
Arising troubles which haven't occurred before with several chemicals or foods are common with those suffering from Systemic Yeast Infection Symptoms.

3. Problems with the gastrointestinal tract.

On many occasions, as touched on in the previous detail of the food allergies, flatulence, inflammatory bowel disease, rectal itching, constipation, and diarrhea are the most common of the Symptoms.

It is even possible for thrush, a Yeast Infection affecting the mouth and/or throat, to develop.

4. Onset of urinary and genital problems.

5. Development of hives and skin rashes.

You could even be having a case of hives, and not know where they came from.

6. Suddenly feeling irritable or mildly depressed.

Many times people complain of Systemic Yeast Infection Symptoms that include: mental confusion, feeling of being in a 'fog', difficulty focusing or concentrating, sleepness nights, memory loss, and decreased attention span.

7. Problems with the autoimmune system.

Some autoimmune disorders that normally become worse from a Systemic Yeast Infection are sarcoidosis, scleroderma, myasthenia gravis, arthritis, hemolytic anemia, thrombocytopenic purpura, or systemic lupus erythematosus.

Yeast flourish on a body that is fed with sugar, refined starch, and chemical additives.
These chemicals, starches and sugars are also readily present in bread, cookies, chips and other junk foods which are a large part of many peoples diets.

When someone is under immense stress, the microorganisms in the body that control the yeast start to die off.

Anytime you think you may have a Systemic Yeast Infection Symptoms, you need to check with your doctor about what tests need to be run to determine what your body's level of Yeast organism is.
All in all, remember that it's vital to know if you are dealing with a Systemic Yeast Infection Symptoms so you can get the proper treatment.

About the author:

Source: http://www.sooperarticles.com/health-fitness-articles/women-health-articles/systemic-yeast-infection-symptoms-7-signs-watch-out-464763.html


symptoms of systemic lupus erythematosus sle

11 thoughts on “Symptoms Of Systemic Lupus Erythematosus Sle

  1. tor

    Is there a diet for those with Lupus?
    I want to know if there are any diets to followto relieve the symtoms of lupus. I heard that citrus aggreivates the joints, which sucks cause I love oranges and lemons. Althafa sprouts are not good either. Anyone have idea where to find a hit list of things I should limit or not eat?
    Are there certain things I should avoid?

    1. kimchungtran

      Dear Asker!

      Here is a good Diet and Lifestyle changes for patients with Systemic Lupus Erythematosus

      The following are some tips for maintaining a healthy diet:

      * Eat a diet low in saturated fats. Not all fats are unhealthy. Some studies suggest that omega-3 fatty acids, which are fat compounds found in fish oil, black currant or primrose seed oils, and flax seed, have anti-inflammatory and nerve protecting actions.

      Omega-3 fatty acids are essential acids. Medical research suggests they may have anti-inflammatory properties.

      * Choose whole grains and fresh vegetables and fruits. According to some studies, a diet rich in fruits and vegetables can lower homocysteine levels, which are elevated in patients with SLE and may be a risk factor for heart disease. Researchers are also investigating compounds called indoles, also known as mustard oil, which are found in broccoli, cabbage, Brussels sprouts, cauliflower, kale, kohlrabi, collard and mustard greens, rutabaga, turnips, and bok choy. Indoles stimulate enzymes that convert estrogen to a more benign type. Eating vegetables certainly will not cure SLE, but they offer many general health benefits.

      * Get most proteins from vegetables, particularly soy.
      * Avoiding dairy and meat products may help protect the kidneys.
      * Take extra calcium and vitamin D to prevent osteoporosis (this is particularly useful for patients taking corticosteroids).
      * Supplements of vitamins B12, B6, and folate may be necessary, especially in people whose blood tests show high levels of homocysteine.
      * Exercise is safe, but patients should not expect it to improve symptoms, including joint aches and fatigue.
      * Restrict salt (particularly for patients with signs of high blood pressure and kidney disease).

      Of possible interest to patients with SLE is a 2002 report that patients with rheumatoid arthritis (also an inflammatory autoimmune condition) experienced improvement when they went on the Mediterranean diet, which stresses fish (which contains anti-inflammatory factors), olive oil, garlic, whole grains, nuts, and fresh fruits and vegetables. In any case, such a diet is heart-healthy, which is important for patients with SLE

      Prevention Against Infections

      Patients should minimize their exposure to crowds or people with contagious illnesses. Careful hygiene, including dental hygiene, is also important.

      Avoiding SLE Triggers

      Simple preventive measures include avoiding overexposure to ultraviolet rays and wearing protective clothing and sunblocks. There is some concern that allergy shots may cause flare ups in certain cases. Patients who may benefit from them should discuss risks and benefits with an SLE specialist. In general, patients with SLE should use only hypoallergenic cosmetics or hair products.

      Reducing Stress

      Chronic stress has profound physical effects and influences the progression of SLE. According to one study, patients with SLE differ from healthy individuals in their immune responses to stress, and psychological stress can induce flare-ups in patients with SLE. Patients should try to avoid undue emotional or physical stress. Getting adequate rest of at least 8 hours and possibly napping during the day may be helpful. Maintaining social relationships and healthy activities may also help prevent the depression and anxiety associated with the disease.

      Hope that may help you!
      Good luck!

  2. Unknown

    What are some of the medical treatments for lupus? How do people die from lupus do their organs sweal in?
    the body how does it happen and what are some of the treatments for lupus?

    1. shygirlxoxo

      Test Overview

      An antinuclear antibody (ANA) test measures the amount and pattern of abnormal antibody that work against the body’s tissues (autoantibody). Everyone has a small amount of autoantibody, but about 5% of people have a larger amount. About half of these 5% have an autoimmune disease, such as systemic lupus erythematosus or rheumatoid arthritis. An ANA test alone cannot diagnose a specific disease. It is used in combination with an evaluation of symptoms and other tests.

      The body’s immune system normally attacks and destroys foreign substances such as bacteria and viruses. However, in disorders known as autoimmune diseases, the immune system attacks and destroys the body’s normal tissues. When a person has an autoimmune disease, the immune system produces antibodies that attach to the body’s own cells as though they were foreign substances, often causing them to be damaged or destroyed. A thorough medical history, physical examination, and other tests besides an ANA test are needed confirm a suspected autoimmune disease.

      Why It Is Done

      A test for antinuclear antibodies (ANA) is done to help diagnose conditions that include:

      Systemic lupus erythematosus (SLE).
      Rheumatoid arthritis
      Sjögren’s syndrome.
      Scleroderma.
      Hashimoto’s thyroid disease.
      A reaction to a medication.

  3. karl j

    What r canine lupus symptoms, what is prognosis. Need to add to the Vets knowledge hopefully?
    my Boston Terrier/Shiz-tsu is at vets he is unable to locate cause of flucuating fever, some drooling, lack of appetite.White blood cell count at the bottom of the scale. All other tests (kidneys, Liver) coming back normal. Was being treated to some infection in esophogus being given anti-biotics. Would like to offer vet an alternative diagnosis

    1. Pam

      Systemic Lupus Erythematosus(SLE): is one of several diseases known as “the great imitators” because its symptoms vary so widely it often mimics or is mistaken for other illnesses, and because the symptoms come and go unpredictably. Diagnosis can be elusive, with patients sometimes suffering unexplained symptoms and untreated SLE for years. Common initial and chronic complaints are fever, malaise, joint pains, myalgias and fatigue.

      Discoid Lupus symptoms: normally starts as loss of pigment around the nose. There may be scabby sores or just scaling of the nasal tissue. The surface of the nose may change from its typical cobblestoned appearance to a smooth surface.

  4. sekhmet179

    What are the four stages of treatment for lupus?
    I remember reading somewhere that there were four stages of treatment for systemic lupus erythematosus I think it was like
    I-corticosteroids
    II-?
    III-?
    IV-Immunosuppressors, chemotherapy, etc

    I was wondering if there was anything like this on the internet or whether I am confusing it with the kidney affected stages? (I doubt it but you never know…)

    Thanks in advance for any info, and please be sure to set up a link to where you got your info

    1. Cheryl M

      What is the treatment for systemic lupus?

      There is no permanent cure for SLE. The goal of treatment is to relieve symptoms and protect organs by decreasing inflammation and/or the level of autoimmune activity in the body. Many patients with mild symptoms may need no treatment or only intermittent courses of antiinflammatory medications. Those with more serious illness involving damage to internal organ(s) may require high doses of corticosteroids in combination with other medications that suppress the body’s immune system.

      Patients with SLE need more rest during periods of active disease. Researchers have reported that poor sleep quality was a significant factor in developing fatigue in patients with SLE. These reports emphasize the importance for patients and physicians to address sleep quality and the effect of underlying depression, lack of exercise, and self-care coping strategies on overall health. During these periods, carefully prescribed exercise is still important to maintain muscle tone and range of motion in the joints.

      Nonsteroidal antiinflammatory drugs (NSAIDs) are helpful in reducing inflammation and pain in muscles, joints, and other tissues. Examples of NSAIDs include aspirin, ibuprofen (Motrin), naproxen (Naprosyn), and sulindac (Clinoril). Since the individual response to NSAIDs varies among patients, it is common for a doctor to try different NSAIDs to find the most effective one with the fewest side effects. The most common side effects are stomach upset, abdominal pain, ulcers, and even ulcer bleeding. NSAIDs are usually taken with food to reduce side effects. Sometimes, medications that prevent ulcers while taking NSAIDs, such as misoprostol (Cytotec), are given simultaneously.

      Corticosteroids are more potent than NSAIDs in reducing inflammation and restoring function when the disease is active. Corticosteroids are particularly helpful when internal organs are involved. Corticosteroids can be given by mouth, injected directly into the joints and other tissues, or administered intravenously. Unfortunately, corticosteroids have serious side effects when given in high doses over prolonged periods, and the doctor will try to monitor the activity of the disease in order to use the lowest doses that are safe. Side effects of corticosteroids include weight gain, thinning of the bones and skin, infection, diabetes, facial puffiness, cataracts, and death (necrosis) of large joints.

      Hydroxychloroquine (Plaquenil) is an antimalarial medication found to be particularly effective for SLE patients with fatigue, skin, and joint disease. Side effects include diarrhea, upset stomach, and eye pigment changes. Eye pigment changes are rare, but require monitoring by an ophthalmologist (eye specialist) during treatment with Plaquenil. Researchers have found that Plaquenil significantly decreased the frequency of abnormal blood clots in patients with systemic SLE. Moreover, the effect seemed independent of immune suppression, implying that Plaquenil can directly act to prevent the blood clots. This fascinating work highlights an important reason for patients and doctors to consider Plaquenil, especially for those SLE patients who are at some risk for blood clots in veins and arteries, such as those with phospholipid antibodies (cardiolipin antibodies, lupus anticoagulant, and false positive VDRL). This means not only that Plaquenil reduces the chance for reflares of SLE, but it can also be beneficial in ‘thinning’ the blood to prevent abnormal excessive blood clotting.

      For resistant skin disease, other antimalarial drugs, such as chloroquine (Aralen) or quinacrine, are considered, and can be used in combination with hydroxychloroquine. Alternative medications for skin disease include dapsone and retinoic acid (Retin-A). Retin-A is often effective for an uncommon wart-like form of lupus skin disease. For more severe skin disease, immunosuppressive medications are considered as below.

      Medications that suppress immunity (immunosuppressive medications) are also called cytotoxic drugs. Immunosuppressive medications are used for treating patients with more severe manifestations of SLE with damage to internal organ(s). Examples of immunosuppressive medications include methotrexate (Rheumatrex, Trexall), azathioprine (Imuran), cyclophosphamide (Cytoxan), chlorambucil (Leukeran), and cyclosporine (Sandimmune). All immunosuppressive medications can seriously depress blood cell counts and increase risks of infection and bleeding. Other side effects are peculiar for each drug. For examples, Rheumatrex can cause liver toxicity, while Sandimmune can impair kidney function.

      In recent years, mycophenolate mofetil (Cellcept) has been used as an effective medication for lupus, particularly when it associated with kidney disease. Cellcept has been helpful in reversing active lupus kidney disease (lupus renal disease) and in maintaining remission after it is established. It’s lower side effect profile has advantage over traditional immune suppression medications.

      In SLE patients with serious brain or kidney disease, plasmapheresis is sometimes used to remove antibodies and other immune substances from the blood to suppress immunity. Some SLE patients can develop seriously low platelet levels, thereby increasing the risk of excessive and spontaneous bleeding. Since the spleen is believed to be the major site of platelet destruction, surgical removal of the spleen is sometimes performed to improve platelet levels. Other treatments have included plasmapheresis and the use of male hormones. Plasmapheresis has also been used to remove proteins (cryoglobulins) that can lead to vasculitis. Endstage kidney damage from SLE requires dialysis and/or a kidney transplant.

      Most recent research is indicating benefits of rituximab (Rituxan) in treating lupus. Rituximab is an intravenously infused antibody that suppresses a particular white blood cell, the B cell, by decreasing their number in the circulation. B cells have been found to play a central role in lupus activity, and when they are suppressed, the disease tends toward remission.

      At the 2007 national Rheumatology meeting, there was a paper presented suggesting that low dose dietary supplementation with omega-3 fish oils could help patients with lupus by decreasing disease activity and possibly decreasing heart disease risk.

    1. Mξ∫ĭŊĐд

      ANTI-INFLAMMATORY AND IMMUNOSUPPRESSIVE ACTIVITIES
      Korean scientists found that pycnogenol had anti-inflammatory and
      immunosuppressive activities, as pycnogenol showed inhibitory effects on
      the expression of the proinflammatory cytokine IL-1 by regulating
      redox-sensitive transcription factors. [1]

      ANTI-OXIDATIVE ACTIVITIES Pycnogenol was found to have strong
      free radical-scavenging activities against reactive oxygen and nitrogen
      species. [2] In a study, pycnogenol prevented vascular endothelial cells
      from injury induced by an organic oxidant t-butyl hydroperoxide and
      inhibited macrophage oxidative burst, plasma low density lipoprotein
      (LDL) oxidation, and hydroxyl radical-induced plasmid DNA damage. [3]
      When, pycnogenol was added to cigarette filters and free radicals were
      found to be depleted in a dose dependent manner. [4]A significant
      increase in oxygen radical absorbance capacity (ORAC) was observed in
      plasma throughout a pycnogenol supplementation period. The average
      melasma area of 30 patients was found decreased by 25.86 +/- 20.39
      mm and the average pigmentary intensity decreased by 0.47 +/- 0.51
      unit after pycnogenol supplementation (25 mg, 3 times a day) for 30
      days. The general effective rate was 80%. [5] Pycnogenol chewing gums
      significantly reduced gingival bleeding and led to no increases in plaque
      accumulation in a double-blinded study. [6] Actually, most of pycnogenol
      benefits found are related to its anti-inflammatory and anti-oxidant
      activities.

      SYSTEMIC LUPUS ERYTHEMATOSUS Pycnogenol treatment reduced
      the reactive oxygen species production, apoptosis, p56(lck) specific
      activity and erythrocyte sedimentation rate and improved SLE disease
      activity index in systemic lupus erythematosus (SLS). [7]

      ASTHMA Asthma is considered as a chronic inflammatory process.
      Because of pycnogenol’s anti-oxidant and anti-inflammatory activities, it
      may have benefits on asthma.

      In a study, patients were assigned to receive either 1 mg/lb/day
      (maximum 200 mg/day) Pycnogenol or placebo for the first period of 4
      weeks and then crossed over to the alternate regimen for the next 4
      weeks. All 22 patients responded favorably to Pycnogenol in contrast to
      placebo. Pycnogenol also significantly reduced serum leukotrienes. [8]

      In an another study, pycnogenol significantly improved pulmonary
      functions and asthma symptoms in 60 subjects aged 6-18 years old,
      suffered from mild-to-moderate asthma. The subjects were able to
      reduce or discontinue their use of rescue inhalers more often than the
      placebo group. There was also a significant reduction of urinary
      leukotrienes in the Pycnogenol group [9].

      CANCER / TUMOR Concentrations of Pycnogenol of 0.05-0.2% was
      applied to the irradiated dorsal skin of kh:hr hairless mice immediately
      after exposure resulted in dose-dependent reduction of the inflammatory
      sunburn reaction. Pycnogenol lotions applied postirradiation also reduced
      this immunosuppression . Chronic exposure to UV on 5 days/week for 10
      weeks induced skin tumors from 11 weeks in both control mice and in
      mice receiving daily applications of 0.05% Pycnogenol, but tumor
      appearance was significantly delayed until 20 weeks in mice receiving
      0.2% Pycnogenol. Consequently, they concluded that topical Pycnogenol
      offered significant and dose-dependent protection from UV-induced acute
      inflammation, immunosuppression and carcinogenesis, when applied to
      the skin after daily irradiation.[10]

      A research group compared the response of human breast cancer cells
      (MCF-7) and normal human mammary cells (MCF-10) to apoptosis in the
      presence of pycnogenol. They plated out MCF-7 and MCF-10 cells in
      culture dishes and grown in medium containing 0, 40, or 80 micrograms
      pycnogenol/ml culture medium. They detected the apoptosis by
      morphology, chromatin condensation, nuclear DNA fragmentation, DNA
      strand breakage or apoptotic bodies. They found that DNA fragmentation
      was significantly higher in MCF-7 cells treated with pycnogenol than the
      untreated cells while pycnogenol did not significantly alter the number of
      apoptotic cells in MCF-10 samples. In conclusion, pycnogenol selectively
      induced death in human mammary cancer cells (MCF-7) and not in normal
      human mammary MCF-10 cells. [11]

      In a mice study, found that pycnogenol was found to have no
      antagonizing effect on the anticancer activity of doxorubicin and
      cyclophosphamide. Pycnogenol possesses a protective effect on the
      cardiotoxicity of doxorubicin and the inhibition of thymus DNA synthesis
      induced by cyclophosphamide in mice. [12]

      Pyconogenol is a strong anti-oxidant; it may also benefit on conditions
      such as thrombosis, platelet aggregation and retinopathy.

      VENOUS THROMBOSIS, THROMBOPHLEBITIS, PLATELET
      AGGREGATION
      In a study, pycnogenol inhibited NF-kappa B activation, VCAM-1 and
      ICAM-1 expression. Thus, pycnogenol might play an

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